Improve LDL-cholesterol levels
and heart health!
Multiple levels of compounding Biological Activity to improve LDL-cholesterol levels and heart health!
CURCUMIN-LDL45TM is engineered curcuminoid technology shown to help manage LDL-cholesterol with similar pharmacological mechanism to statin drugs. This technology not only lowers serum LDL cholesterol it reduces oxidative stress associated with LDL oxidation and inhibits inflammation associated with atherosclerotic plaque formation – a synergistic THREE pronged attach against cholesterol mismanagement and heart disease.
Engineered curcuminoid technology is shown to help manage LDL-cholesterol with a similar pharmacological mechanism to statin drugs. Curcumin extracts typically deliver a curcuminoid proportion as: diferuloylmethane (curcumin I) 75–80%; demethoxycurcumin (DMC — curcumin II) 9-12%; and bisdemethoxycurcumin (BDMC — curcumin III) 0.2–2%. Regular curcumin extracts (even the 95% variation) maximally delivers 12% of this activity and therefore are weak with regards to this cholesterol-correcting activity. The patented curcumin LDL45™ is engineered to deliver a minimum 45% concentration of these cholesterol-correcting curcuminoids.
CURCUMIN LDL45™ BENEFITS:
Helps scavenge serum LDL
Helps reduce LDL synthesis
Extremely potent anti-oxidant activity to protect serum LDL from oxidation (LDL oxidation sets in motion atherosclerotic plaque development)
Anti-inflammatory activity to help mitigate immune-mediated advancement of plaque development
Provides antioxidants for the maintenance of good health. Used in herbal medicine to help relieve joint inflammation.
Adults: 1-3 Capsules daily.
Cautions and warnings: Consult a health care practitioner prior to use if you are pregnant or breastfeeding, if taking antiplatelet medication or blood thinners; if suffering from bile duct obstruction such as gallstones; if you have stomach ulcers or excess stomach acid; or if symptoms persist or worsen. Do not use if seal on cap is broken or missing.
HMGCoA is an enzyme that performs at a critical point in the pathway responsible for the synthesis of LDL cholesterol in our cells to make LDL abundantly.
Curcumin II most effectively (with greater significance) REDUCES the enzyme levels to result in reduction of LDL cholesterol manufacturing.
LDL Receptor Expression
LDL Receptor is cell surface receptor responsible for collecting free flowing LDL cholesterol from blood circulation and bringing into the liver for destruction or removal.
Curcumin II effectively INCREASES synthesis of these scavenging receptors resulting in a second mechanism that lowers serum LDL cholesterol.
Clearing the Path to Potent Longevity
Attain Healthy Essential Fatty Acid Status and Healthy Inflammatory Activity.
E-BOOK EXCERPT: CURCUMIN— A POWERFUL ANTI-INFLAMMATORY
Curcumin displays strong anti-inflammatory potential in the entire body, and in the near future it will be recognized as one of the vital anti-aging nutrients. It plays a core role in any health program. I’ve been using extremely high doses of curcumin (turmeric 95% curcuminoids) for various reasons, which you’ll see disclosed throughout this book series.
Curcuminoids, extracted from turmeric, contribute a potent defense of DNA. Studies have compared its anti-inflammatory potential and
anti-cancer properties against common pharmaceutical therapies to reveal, in some cases, better results and in other cases conclusive synergistic value. Have pharmaceutical companies reported on this? No, they haven’t. The incredible facts rising out of this research reveal that curcumin, unlike a common nonsteroidal anti-inflammatory drug (NSAID), doesn’t induce gastrointestinal, liver, and/or kidney toxicity.
Having read reports of the potential of curcuminoids to down-regulate
cyclo-oxygenase-enzyme activity and eicosanoid-hormone biosynthesis, I was confident some time ago that they regulated gene expression of the enzymes involved in eicosanoid-hormone production, much like the protective NF-kappa-B gene influence of alpha lipoic acid. In this way curcuminoids prevent the overexpression of genes involved in the immune and inflammatory response.
After ongoing, relentless search, I found my answer. Curcumin had
been investigated for its ability to suppress the activation of HIV to the status of AIDS. In a 1993 Harvard University study it was demonstrated that curcumin inhibited the expression of a gene in the DNA of the virus. This gene was responsible for turning on HIV to initiate the assault on the body. Recently, through this historical work, curcumin has been shown to block the activation of NF-kappa-B. Not only is curcumin a powerful HIV inhibitor, it can be justifiably applied as part of the anti-aging and anti-inflammatory program with scientific proof that it works powerfully alongside or independent of alpha lipoic acid. This insight, was the impetus for my push to study curcuminoids exclusively and define with more accuracy how they can be used and even engineered to increase their potential to bring general health benefits and disease treatment.